Eczema: Stop The Itch
Eczema (also known as atopic dermatitis) is a general term used to describe a strange variety of skin rashes ranging from small sections of skin that are slightly itchy, dry and irritated to chronically inflamed, oozing, crusted areas covering the entire body and accompanied by incessant itching. Eczema can have multiple appearances, looking and feeling completely different person to person. The most common areas for eczema to occur are in the folds of the arms and legs, the back of the neck, back of the hands, tops of feet, and the wrists.
In This Article:
What Causes Eczema
One of the predominant theories is that someone with eczema has a short-circuited immune response where skin reacts abnormally when a substance comes in contact with it. In severe cases of eczema, the substance can be as benign as water. For others the trigger can be anything from clothing, detergents, soaps, grass, food products, allergens (including dust mites) to a lack of humidity, or a combination of elements. Even more frustrating is that the reaction can be intermittent with no real rhyme or reason for why or when.
There also appears to be a hereditary component to eczema. For example, children whose parents suffer from eczema run an 80% chance of developing it themselves. Further, in both children and adults, stressful situations tend to trigger, prolong, or worsen eczema flare-ups.
Regardless of the source, eczematous skin reacts to a substance or substances or environmental conditions by spinning out of control and generating mild to severe inflammation, which produces itching and scratching.
Types of Eczema
Atopic eczema (also referred to as atopic dermatitis): Perhaps the most pernicious and painful types of eczema, it's characterized by its severity and intolerable sensation of itching and irritation leaving skin raw, fissured, and vulnerable to infection.
Allergic or irritant contact dermatitis: This specific form of eczema occurs when a particular substance comes in contact with the skin causing the immune system to overreact, becoming inflamed and sensitized. Most typically this can be caused by fragrance, nickel, detergents, wool, grass, citruses, household cleaning products, and vinegar. Once you’ve identified the specific substance, avoiding it often solves the problem. A subset of this condition is eyelid dermatitis. Typically mild to moderate redness is present, as well as scaling, flaking, swollen skin. This is extremely common and almost exclusively affects women in relation to their use of hairstyling products, makeup, and nail polish when it comes in contact with the eye area with the most common culprits being nail polish, perfume, and preservatives. The best way to solve the problem is to stop use of the offending product(s) and find options that don’t trigger a reaction.
Infantile seborrhoeic eczema: Better known as cradle cap, this form of eczema generally only affects babies during the first year of their life. The crusty, thick, sometimes reddened lesions may look problematic, but this disorder is rarely itchy or even felt by the child.
Adult seborrhoeic eczema: Shows up for most people past the age of 20 and 40. It is usually seen on the scalp as mild dandruff, but can spread to the face, ears and chest. The skin becomes red, inflamed and starts to flake. The condition is believed to be caused by a yeast growth. If the condition becomes infected, treatment with an anti-fungal cream may be necessary.
Nummular eczema: Typically localized on the legs, nummular eczema is characterized by coin-shaped patches of pink to red skin that may take on an orange cast if crusting or scaling is present. If treatment is not used, the dry, scaly spots typically darken and thicken. This type of eczema is most common in adolescent girls and women between the ages of 50–60 and the condition tends to occur in winter.
With no cure on the horizon, the good news is that there are still a number of treatments that can help reduce symptoms and mitigate the level of discomfort eczema can cause.
Gentle, effective skin care: The first line of defense is a gentle, fragrance-free skin-care routine that prevents or reduces inflammation and keeps the skin moist and its barrier intact. Improving the skin’s outer structure by providing it with antioxidants, ingredients that improve the skin’s barrier, anti-irritants, and emollients can offer amazing results for most forms of eczema.
Avoid irritants: Aside from using a gentle skin-care routine and a well-formulated moisturiser, avoiding the things that can trigger skin reactions is also of vital importance. Steering clear of known irritants and prolonged contact with water can be incredibly beneficial. For your hands, it also helps a great deal to reapply moisturiser within seconds of washing any part of the body, but especially the hands because soaps and cleansers are notorious for triggering a reaction in those struggling with eczema.
Topical steroids: The most typical and successful medications used are prescription-strength topical steroids (cortisone creams). Over-the-counter cortisone creams can be effective for very mild or transient forms of eczema but when those fail, prescription cortisone creams can save your skin. Though there are no short-term detrimental side effects of using most strengths of cortisone cream, it is still important to only apply it on the affected areas and then only as needed. Repeated, prolonged application of cortisone creams can cause thinning of the skin and prematurely age it.
A Gentle Combination: Paula's Choice Skin Care products for eczema-prone skin takes into account the use of prescription or over-the-counter products to assist in dealing with this skin condition. These therapies will be complemented by our gentle cleanser, soothing toner, protective sunscreen, and state-of-the-art moisturisers. All of our products are filled with effective, potent antioxidants and barrier repair ingredients missing from lots of products claiming to provide relief of eczema-prone skin.
Oral steroids: In severe cases of eczema when topical steroids have failed to produce any relief, oral steroids may be prescribed, but only under a doctor's scrutiny due to the serious side effects associated with this type of medication.
Topical Immunomodulators: In 2000 and 2001, The American FDA approved new topical drugs for the treatment of eczema known as immunomodulators. These are not cortisones or steroids but drugs that can regulate the skin's immune response. Regrettably, in March 2005, the FDA announced a public health advisory for these drugs "to inform healthcare providers and patients about a potential cancer risk..." The FDA said that the risk is uncertain and advises that these drugs should be used only as labeled, for patients who have failed treatment with other therapies. (Source http://www.fda.gov/cder/drug/advisory/elidel_protopic.htm.)
Phototherapy: Research has shown that exposing skin to UVA or UVB light can help reduce the symptoms of chronic eczema. Under medical supervision the use of specially designed bulbs encased in a box can allow affected parts of the body to be exposed to the light source. More severe or chronic eczema can be treated with UVA light in combination with a prescription medication called psoralen. Psoralen can be administered either orally or topically, increasing the skin’s sensitivity to light. This treatment is known as PUVA (Psoralen + UVA light).
Phototherapy treatments are complicated and expensive. They are administered several times per week over a span or one week to several months at a doctor’s office. Moreover, the risk of accelerated ageing of the skin and increased risk of skin cancer from ultraviolet radiation therapy can be the same as for sunbathing.
For those looking for alternatives to prescription treatments and methods over and above the ones mentioned above, an interesting study on eczema appeared in the January 2001 issue of The Archives of Dermatology. This study reports research in Japan that demonstrated that two-thirds of the patients with eczema improved after a month of drinking a liter of oolong tea daily. According to the study "118 patients … were asked to maintain their dermatological treatment. However, they were also instructed to drink oolong tea made from a 10-gram teabag placed in 1000 milliliters of boiling water and steeped for 5 minutes. After 1 month of treatment 74 (63%) of the 118 patients showed marked to moderate improvement of their condition. A good response to treatment was still observed in 64 patients (54%) at 6 months."
The study concluded that "The therapeutic efficacy of oolong tea may well be the result of the anti-allergic properties of tea polyphenols." Whilst the study didn't look at the effect of tea drinking if the topical treatments were stopped, the patients did receive some benefit. So by combining topical treatments (moisturisers and possibly cortisone cream) with some oolong tea, perhaps the benefits will add up so those with eczema can breathe a sigh of relief.
Evening primrose oil and borage oil contain gamma linolenic acid, which may play a part in general skin health and has gained a reputation for reducing occurrences of eczema when applied topically. Several studies, including one in the British Medical Journal (December 2003, pages 1358-1359), have shown that not to be the case. Nonetheless, if you are interested in alternative treatments for eczema this is one you can try with very little risk of adverse effects to see if it may work for you.
There is also research pointing to dietary considerations as being a source of reactions. It is worth experimenting to see if eliminating certain food groups such as dairy, gluten, processed foods, or nuts can improve the severity or frequency of outbreaks you experience.
Sources: Dermatologic Therapy, September 2004, page 264; Pediatrics, January 2006, pages 118-128; Current Medical Research and Opinion, November 2005, pages 1735-1739; British Journal of Community Nursing, October 2005, pages 453-456; Homeopathy, October 2005, pages 215-221; Pediatric Allergy and Immunology, September 2005, pages 527-533; American Journal of Clinical Dermatology, June 2005, pages 203-213; Journal of Dermatology, May 2005, pages 346-353; British Journal of Dermatology, June 2005, pages 1193-1198; Skin Pharmacology and Physiology, May-June 2005, pages 103-114; Archives of Dermatology, December 2004, pages 1463-1466; and The Skin Sourcebook, Alan S. Boyd, M.D., 1998, pages 45-62.